What is cancer?
Cancer begins to work when the body cells are constantly produced but the body cannot control, leading to the formation of new cells with abnormal expressions. These abnormal cells form large arrays, clots and often called tumors.
What is cancer, why is it hard to prevent?
If tumor cells do not spread and develop, known as benign, they are not cancer and can often be eliminated. Conversely, if the cells can penetrate into the tissue or healthy organs, or spread throughout the body through the blood or lymph system, and constantly thrive, these tumors will be called malignant tumors or cancer. At this point, the cancer cells will spread faster if tumors are untreated.
What causes cancer?
Each cell in the human body contains DNA sequences (short for deoxyribonucleic acid phrase – genetic material in humans and almost all living organisms). DNA carries the genetic code of living organisms and performs operations as directed by the cells.
If the DNA in the cell is damaged, the operating instructions will be wrong. In fact, the DNA damaged or suddenly mutated constantly occurs in the cells of a living body when cells divide or reproduce. The majority of the cells are able to recognize when mutations occur, then self-repair the DNA sequences or self-destruct and die.
When the number of mutations occurring in the DNA of a cell is just enough, functional control of cell growth may be lost but the cells do not die. Instead, they begin to develop under the abnormal guidance making them reproduce and develop, produce more and more mutated cells – this is the initial stage of cancer.
Many factors, such as smoking or exposure to sunlight, are the cause of DNA damage – accumulating mutations lead to cancer faster. In addition, a family history of cancer also increases the risk of serious diseases, because you are born a human inherited some DNA mutations that might cause cancer.
Even if the illness is in remission, the cancer is still very easy to grow back. However, in many cases, scientists have yet to figure out the exact causes or chains of events causing cancer.
A recent study has found that more than 80 genetic markers (for example mutations) might increase the risk of developing cancers such as breast, prostate, or ovarian cancer. Scientists believe that these studies may soon be used as a basis for the widespread application of DNA testing for diagnosis and treatment overflow variants of cancer.